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For a prisoner with membranous nephropathy who is being treated with rituximab, is isolation required solely because the patient received rituximab? There is no clear local regulation for that Your advice is appreciated
I think this is genetic FSGS in both siblings. The absence of hypoalbuminemia makes Primary FSGS very unlikely. About 10-15% of patients with genetic FSGS will show a reduction of proteinuria with a CNI, due to a direct stabilizing effect on podocytes ...
I agree this is secondary FSGS based on pathology and on no edema Would have predicted a COL4 a3-5 but that is not supported by the genetic studies, and how can you ignore the podocin gene abnormalities in both sibs? I think the CNI lowering of ...